These results showed that the energy metabolism dysfunction was the initial stage, and the damage of oxygen free radicals was the major factor in ischemia-reperfusion injury.
这些结果说明线粒体能量代谢障碍是缺血再灌注损伤的始动环节，而氧自由基是造成损伤的主要因素。
2
OBJECTIVE To investigate the protective mechanism of adenosine in renal damage of ischemia reperfusion.
探讨腺苷对肾脏缺血再灌注损伤的保护作用机制。
3
By the time of reperfusion, fetal brain damage increases progressively.