BACKGROUND Increasing the activity of defective cystic fibrosis transmembrane conductance regulator (CFTR) protein is a potential treatment for cystic fibrosis.
背景提高有缺陷的囊性纤维化跨膜电导调节(CFTR)蛋白的活性是囊性纤维化的一种潜在治疗。
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Genistein, a naturally occurring isoflavone, augments in vitro epithelial anion transport via activation of the cystic fibrosis transmembrane conductance regulator chloride channel.