Conclusion: GAP-43 and ET-1 play important roles in the pathophysiological process of blood brain barrier damage after diffuse brainlesion.
结论GAP - 43及ET - 1在脑损伤后血脑屏障损害的病理生理过程中起重要作用。
2
There were no relationship between mirror-writing and the time procedure of disease. Mirror-writing was not a transitional phenomenon of brainlesion;
镜像书写与病程无关，其并非为脑损伤后的、一过性的暂时现象；
3
MethodS: a model of ischemia-reperfusion induced brainlesion was set up by ligation of common carotid arteria in rats, and the protective effects of THP was observed.