Caffeine is a pro at mimicking a neurotransmitter called adenosine.

咖啡因是一种能够像“腺苷”神经递质一样在神经细胞间传递信息。

In fact, caffeine works by blocking adenosine's receptor pathways.

事实上，咖啡因是靠阻断接受腺苷的感知器官来实现其作用的。

But because it's not adenosine, the" sleepiness" effect isn't felt.

但是，由于咖啡因不是腺苷，所以再怎么积聚你都不会感觉到有睡意。

Firstly, caffeine blocks the receptors in the body for a compound called adenosine.

首先，咖啡因会阻断体内对腺苷化合物的受体。

There are four bases: adenosine, thymine, cytosin, and guanine.

腺苷、胸腺嘧啶、胞嘧啶和鸟嘌呤。

We gave him 40 of furosemide. Rhythm didn't change after 6 of adenosine.

我们给他注射40个单位利尿剂。在注射了6个单位腺瞟呤核甘后心律不变。

No, no, no, it only stops your heart for six seconds...-Meredith, it's adenosine.

不，不，不，这个只会停住你心跳6秒... -Meredith，是腺苷。

So caffeine stops adenosine from doing its thing, while some stimulating neurotransmitters are still around.

所以说咖啡因抑制了腺苷酸执行它的职责，然而那些兴奋的神经传导物质依然在周围。

So the caffeine doesn't even need to compete with the adenosine to fit into those receptors.

所以咖啡因不用和腺嘌呤核苷竞争受体。

As adenosine builds up, it increases the urge to sleep, also known as sleep pressure.

当腺苷累积起来的时候，它增加了困倦感，也就是睡眠压力。