Thrombin also turns factor XIII into factor XIIIa which cross links the fibrin to form a stable clot.

凝血酶也能使因子XIII转变为因子XIIIa；因子XIIIa与纤维蛋白通过交叉连接形成稳固的血凝块。

With coagulase negative species, like Staph saprophyticus or Staph epidermidis, the fibrin doesn't clump up.

对于凝固酶阴性的细菌，例如腐生葡萄球菌或表皮葡萄球菌，纤维蛋白则不会结块。

By inhibiting both the intrinsic and common pathway, less fibrin is produced and coagulation slows down dramatically.

通过抑制内源性和共同凝血途径，纤维蛋白产生减少，凝血速度大大减慢。

Coagulase-positive bacteria, like Staph aureus, convert the soluble fibrinogen into sticky fibrin, which then visibly clumps up.

凝固酶阳性细菌（如金黄色葡萄球菌）能将可溶性纤维蛋白原转化为粘性纤维蛋白，从而出现肉眼可见的团聚现象。

And this process produces fibrin degradation products.

这个过程产生了纤维蛋白的降解产物。

Chronic pericarditis typically results from fibrin deposition in the serous pericardium and it prevents the heart from fully relaxing.

慢性心包炎源于纤维蛋白在浆膜心包沉积，影响心肌的舒张充盈功能。

Usually the clot might start naturally breaking down, for example, enzymes like plasmin break down fibrin into fragments called D-dimers.

通常情况下血栓会开始溶解，比如纤维蛋白溶解酶可将纤维蛋白溶解成D二聚体的片段。

The main role of protein C and protein S is to prevent excess coagulation, or fibrin formation, during secondary hemostasis.

蛋白C和蛋白S的主要作用是在二期止血时防止过度凝血（即纤维蛋白形成过多）。

This slows down blood flow, allowing platelets, clotting factors, and fibrin to stick together.

这会减慢血流速度，使血小板、凝血因子和纤维蛋白粘在一起。

This is where exudate, which contains red blood cells, neutrophils, and fibrin starts filling the airspaces and makes them more solid.

渗出物含有红细胞、中性粒细胞和纤维蛋白，开始填满气腔，使肺实变。