MAPK expressed abundantly in hypertrophic myocardium, mainly localized in interstitial cells.

免疫组化研究显示高血压大鼠心肌内有丰富的MAPK表达, 且主要分布于心肌间质细胞.

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Results: LPA enhanced the cultured VSMC ? 3 H ? TdR incorporation in a concentration? dependent manner, and increased MAPK activity concurrently.

结果: LPA呈浓度依赖地刺激3H? TdR参入, 并平行地激活MAPK,二者间呈显著正相关.

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Treatment with CHEL significantly increased hCG accumulation in JAR cells , and enhanced the phosphorylation of MAPK.

对照组及各实验组都有活性的MAPK的 表达,与对照组相比,PMA慢性 刺激组、CHEL组MAPK磷酸 化水平升高;PMA急性 刺激组MAPK磷酸化水平降低.

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MAPK inhibitor ( PD 98059 ) inhibited the phosphorylation of GSK - 3 P and almost completely blocked oxLDL - induced tubular EMT.

MAPK抑制剂 PD98059 可抑制GSK -3 β的磷酸活化,并几乎完全阻断oxLDL诱导的肾小管上皮细胞转分化,但对LDL诱导 的小管上皮细胞转分化只有部分阻断作用.

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MAPK signaling transduction is a key pathway of cellular proliferation and apoptosis regulation.

MAPK信号转导通路是一条关键的调节细胞增生和凋亡的通路.

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Age - related alteration in hepatic acyl - CoA: cholesterol acyltransferase and its relation to LDL receptor and MAPK.

(与年龄相关的肝酰基辅酶A的改变: 胆固醇酰基转移酶及其与LDL受体和MAPK的关系).

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CONCLUSION: Sodium ferulate inhibits p 38 MAPK activation triggered by A ? ? 25 - 35.

结论: 阿魏酸钠可抑制Aβ25-35介导的p38MAPK活性,使TNF- α、iNOS、 NO水平降低.

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Simvastatin may effectively attenuate DN progression through down - regulating the expression of TGF - ? ? via p 38 MAPK.

斯伐他汀可能通过抑制p38MAPK磷酸化而减少TGF - β的蛋白表达.

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In the YIQIJIEDU granule group, some genes in array were lowly expressed , such as STK 1, CDK , MAPK, et al.

HumanCancercDNAArray分析发现,益气解毒颗粒组有STK1 、 MAPK1 、 CDK1等19个与细胞增殖、细胞周期 、 核转录因子、DNA修复 相关基因表达下调.

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