Serum concentrations of glucose, glycosylated serum protein, and malondialdehyde, derived from lipid peroxidation were also examined to estimate metabolic control.
并检测血糖、糖基化血清蛋白和血清脂过氧化产物丙二醛浓度以反映代谢控制。
2
Inhibiting lipid accumulation in macrophage-derived foam cells and reduce inflammatory responses may be of therapeutic value in preventing coronary artery disease.
抑制巨噬细胞源泡沫细胞中脂质聚积、减轻炎症反应可能有动脉粥样硬化预防、治疗价值。
3
Conclusion: the protective mechanisms of YQTLP were related to inhibit lipid peroxidation, protect endothelium-derived relaxing factor and adjust the fibrinolytic activities.