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Conclusion PKC took place translocation activation during cerebral ischemia reperfusion, PKC promoted the expression of FOS and BCL 2. Their complex role influenced the neuronal apoptosis. 结论缺血再灌注期间PKC发生易位激活，PK C促进了FOS和BCL - 2的表达，这几个因素综合作用影响神经细胞凋亡。 - 2
Analysis of Synaptonemal Complex from a Carrier with 46, XY, t (11; 18) Balanced Translocation. 一位46,xy, t(11； 18)平衡易位携带者的联会复合体分析。 - 3
A Rare Carrier of Complex Translocation With 46, XY, t (4; 21; 11) Causes Habitual Abortion. 一例罕见的46,xy, t(4； 21； 11)复杂易位携带者引起习惯性流产。
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