Objective To study whether nitric oxide (NO) and neuronal NOsynthase (nNOS) were involved in the pathogenesis of acute focal cerebral ischemia and reperfusion.
目的探讨一氧化氮(NO)和神经元型NO合酶(n NOS)是否参与急性局灶性脑缺血再灌注的发病机理。
2
The activities of organic acids-metaboling enzymes showed: There was no relation between the activity of citrate synthase(CS) and organic acids concentration.
有机酸相关代谢酶的活性表现为：柠檬酸合酶（CS）活性变化与有机酸含量相关性不大；
3
Recent researches confirm that the neurons containing NOsynthase can be shown by NADPH-diaphorase histochemical method.