AIM: To investigate the role of phosphoinositide pathway in the formation of pressure-overload cardiac hypertrophy.
目的:探讨肌醇磷脂途径在压力超负荷性心肌肥厚形成中的作用。
2
Suppression of target of rapamycin, a downstream effector of phosphoinositide 3-kinase, also prevented lipofuscin accumulation in the heart.
雷帕霉素作用点(即为磷酸肌醇-3激酶基因下游的受动器)的抑制也同样能够阻止脂褐质在心肌的积聚。
3
Objective to study the expression of phosphoinositide 3 kinase (PI3-K) and its relationship with lung cell apoptosis in rats with acute lung injury induced by oleic acid.