CONCLUSION: the apoptosis of alveolarepithelialcell is induced by high level of endogenous no in the development of pulmonary fibrosis.
结论:在肺纤维化形成过程中,内源性NO的增多,有诱导肺泡上皮细胞凋亡的作用。
2
The implanted MSCs which escaped to the liver, spleen and lung did not differentiate into fibroblast, myofibroblast or alveolarepithelialcell.
同时,我们观察到逃逸到肝脏的移植细胞表达了功能性肝细胞的标志物。
3
RAS inhibition can reduce cell apoptosis of alveolarepithelial cells and endothelial cells, interfere with the inflammation cascade, and decrease fibroblast activity during tissue repair process.