BACKGROUND: brain injury often causes secondary cerebralischemia and hypoxia, which aggravate the brain damages.
背景:脑损伤后常引起继发性脑缺血、缺氧,使脑损害加重。
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Conclusion Propofol may inhibit hypoxia in the brain and the apoptosis of nerve cells in result of protecting the cerebralischemia and reperfusion damage in rats.
结论异丙酚可能通过抑制大脑缺氧,抑制神经元的凋亡,从而对大鼠局灶性脑缺血-再灌注损伤发挥保护作用。
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Objective: Explore the effects of YTG on red cells deformation ability of rat with cerebralischemia and mouse's decompression hypoxia ability.