Previous works have also shown early myelin detachment from the axolemma, but Abrahams et al., who also addressed this issue, did not find this alteration at 24 h after nerve crush.
The axolemma itself, consisting of mostly type 1 and type 2 biological membrane, has been found to be impervious to ions.
Hence the axolemma appears to be the barrier for osmotically induced flow into the axoplasm.
Some cases are due to acute axonal neuropathy, in which glycolipid in the axolemma is targeted.
Myelin sheath of lipoprotein around the axolemma is interrupted at regular intervals to leave the axolemma ` bare’ at nodes of Ranvier.
Origin
Late 19th century: from Greek axōn 'axis' + lemma 'husk'.